This is a simple neuron, so no tricks here. The answers are 1) Soma or cell body; 2) dendrites; 3) the axon; 4) the myelinated sheath; 5) terminal buttons or terminal bulbs

Dendrites receive a neuro-chemical stimulus by having a neurotransmitter attach to a receptor site; the electrical charge that passes through the neuron will then activate the terminal bulbs to release neurotransmitters so that the process continues.

Some neurotransmitters are commonly described as "excitatory" or "inhibitory". The only direct effect of a neurotransmitter is to activate one or more types of receptors. The effect on the postsynaptic cell depends entirely on the properties of those receptors. It happens that for some neurotransmitters (for example, glutamate), the most important receptors all have excitatory effects: that is, they increase the probability that the target cell will fire an action potential. For other neurotransmitters (such as GABA), the most important receptors all have inhibitory effects. There are neurotransmitters, such as acetylcholine, for which both excitatory and inhibitory receptors exist; and there are some types of receptors that activate complex metabolic pathways in the post-synaptic cell to produce effects that cannot appropriately be called either excitatory or inhibitory. Thus, it is an oversimplification to call a neurotransmitter excitatory or inhibitory.

Neurotransmitters must be broken down once they reach the post-synaptic cell to prevent further excitatory or inhibitory signal transduction. There are two ways that this may happen. First, an enzyme may break down the neurotransmitter; for example, acetylcholine is broken down by the enzyme acetylcholinesterase. The second method of elimination of neurotransmitters from the synapse is "re-uptake." Re-uptake is the reabsorption of a neurotransmitter by the terminal buttons of the pre-synaptic neuron. Reuptake allows for the recycling of neurotransmitters; it regulates the level of neurotransmitter present in the synapse and controls how long a signal resulting from neurotransmitter release lasts.

It means that psilocybin has a similar chemical structure to serotonin and is able to fit into serotonin receptor sites, causing the same inhibition of the neuron that serotonin would cause.

Scopolamine fits into the receptor sites of acetylcholine, blocking the ability of acetylcholine to create a memory. This drug has been used both in animal and human research to test the role of acetylcholine.  In most experiments using scopolamine, those receiving the drug are the treatment group that is compared to a control group, where the normal activity of acetylcholine is assumed.